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Anatomy

Style anatomy: Maha Burney – The Express Tribune

The creative director of NFK Photography breaks down her incredible style

The creative director of NFK Photography breaks down her incredible style. Find out all the challenges she faces in dressing her body type and which rules, from her fashion rulebook, she always abides by

Understanding your body is the key to looking good and a trait found amongst all impeccably dressed fashionistas. While people shy away from talking about their bodies, these brave souls explain how they work their anatomies to their advantage

How would you describe your body type?

Small framed.

Has your body type changed over the last five years?

My body has seen the biggest changes over the last five years; from thin, to pregnant, to a squishy mom-body. My post-baby body is definitely a challenge to dress sometimes. Even though I am close to being back to my pre-baby weight, the pounds are all in different places, which means I have to adjust the way I dress.

In your opinion what is your most troublesome area?

Even at my fittest I have always had a tummy. Now, after a c-section it has definitely become my biggest trouble spot!

How has your style changed over the years?

My style has matured with time. I dont experiment as much as I used to in terms of colours or prints and know what suits me and what doesnt. I tend to go for classic, fuss-free, minimal silhouettes, sometimes with a structured edge. Generally I wear a lot of solid colours, but when I do go for prints I like them to either be bold and geometric, or quirky and fun. I like my shoes and bags to be the on-trend statement-making element in my outfit. A statement bag paired with a classic black or white top, skinnies and some beautiful heels is my go-to.

How do you dress according to your body type and which silhouettes suit your body the most?

I think its important to balance your proportions when dressing for your body type, therefore I have a couple of personal dos and donts when it comes to what I think works for me. I like to wear tops that accentuate the length of my torso and lowers that are more tapered and fitted. Neither crop-tops nor culottes suit me, as they cut my torso or lower half off abruptly, and make me look shorter than I am. Also if I am wearing wide-leg pants, they have to be touching the ground as it gives me more length and creates a balance. However, getting dressed isnt always about body type for me. A lot has to do with comfort and my mood in that moment.

In your opinion what is the biggest mistake a person can make while dressing here?

I find it jarring when people wear too many statement pieces all at once without thinking about whether they actually work together.

What is the one piece of clothing you shy away from wearing and why?

Thats an easy question, a swimsuit. I definitely need to hit the gym first!

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Style anatomy: Maha Burney - The Express Tribune

Human Behavior

Here’s Every Moment That Influences Human Behavior – Inverse

Human beings are a mess of contradictions. A person might hate violence but love violent action movies. Someone else might consider certain people are inherently good yet refuse to believe in the idea of a soul. For a long time, scientists have attempted to account for the inconsistencies in human behavior and tried to find predictable patterns in them but theyve yet to come up with a simple explanation.

But neuroscientist Robert Sapolsky wants us to know that were never going to find one.

While we are a miserably violent species, were also extraordinarily compassionate and altruistic, said Sapolsky, a Stanford University professor, to a TED 2017 crowd on Thursday. So how do you make sense of the biology of our best moments, our worst, and all of the ambiguous ones in between?

Answering this, he says, means first accepting that human behavior cant be explained by one hormone or evolutionary mechanism. Human decisions, he explains, are influenced by multiple factors that operate on a vast timeline, ranging from the second before a choice is made to the moment thousands of years ago when a persons behavioral patterns began to be shaped by their ancestors. Heres how he broke down the moments that contribute to human behavior in his talk.

One second before the decision: Whatevers happening in your immediate environment seconds before you make a decision may activate your amygdala, the brain area central to fear and aggression. So, if your environment is stressful, then your amygdala will be more likely to elicit hostile emotions that may influence that choice.

In addition, if youre tired, youre hungry, youre in pain your frontal cortex is not going to work very well, Sapolsky advises. Thats the brain region whose job it is to get there just in time to stop the amygdala.

Hours to days before the decision: This span of time is most influenced by hormones, Sapolsky says. The levels of these natural chemicals constantly fluctuate, and their levels at any given moment can influence a decision made down the line.

Regardless of your sex, if your testosterone levels are elevated, youre more likely to mistake a neutral facial expression for a threatening one, says Sapolsky. Or, if you have elevated levels of stress hormones, your amygdala becomes excitable and your frontal cortex gets sluggish.

Weeks to months before the decision: This time frame is the realm of neural plasticity the changes to the brain that happen as neurons form new connections.

The brain can change dramatically over time in response to experience, says Sapolsky. If your previous months were filled with stress and trauma, your amygdala would have grown larger, and neurons would have grown new connections there.

Years before the decision: The decisions you make as an adult are also shaped by the way your brain forms as it matures during childhood and adolescence, Sapolsky says.

Thats the time that your brain is being constructed, and experiences can cause what are called epigenetic changes, says Sapolsky. Some genes are activated permanently, other ones are turned off. For example, if as a fetus you were exposed to high levels of stress hormones from moms circulation, epigenetic changes would have gifted you with an adult amygdala thats enlarged with elevated stress hormones.

Centuries before you were even born: Ultimately, your behavior and the decisions that arise from that behavior is also shaped by the decisions your ancestors made thousands of years before. For example, Sapolsky explains, if your ancestors were warriors, their attitudes still influence the values with which you are raised.

Its clear that if you want to understand a behavior a wonderful one, an appalling one, a confusing one in between youve got to understand what happened from the second before to millions of years before, Sapolsky said, summarizing his talk.

In other words, its complicated.

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Here's Every Moment That Influences Human Behavior - Inverse

Genetics

Seattle Genetics (SGEN) Q1 Loss Widens, Sales Top Estimates – Yahoo Finance

Seattle Genetics, Inc.SGEN reported a loss of 42 cents per share for the first quarter of 2017, wider than the Zacks Consensus Estimate of a loss of 41 cents and the year-ago loss of 15 cents per share.

Revenues came in at $109.1 million, down 1.8% year over year, primarily due to lower royalty revenues from the sales of Adcetris. Revenues, however, beat the Zacks Consensus Estimate of $103.3 million.

Seattle Genetics share price has gained 29.1% year to date compared with the Zacks classified Medical - Biomedical and Genetics industrys rise of 4.8% in the same period.

Quarter in Detail

Seattle Genetics top line comprises product revenues, collaboration and license agreement revenues and royalties.

The companys only marketed product, Adcetris, generated revenues of $70.3 million, up 20% year over year.

Collaboration and license agreement revenues increased 8.2% to almost $21.8 million. Collaboration revenues included fees earned from the companys agreement with Takeda Pharmaceutical Company Ltd. for Adcetris and other ADC collaborations.

Royalty revenues declined 47.5% year over year to $16.98 million. The decline was due to unfavorable year-over-year comparisons. The company had received a milestone payment of $20 million from Takeda in the first quarter of 2016 on international sales of Adcetris.

Research and development (R&D) expenses were $118.2 million, up 27.3% year over year. Also, selling, general and administrative (SG&A) expenses increased 29.1% to $38.4 million. Costs were high primarily due to investment in vadastuximabtalirine, Adcetris collaboration activities for product supply to Takeda and pipeline development.

In its call, the company asserted that there was no erosion of its share in the relapsed HL business despite the FDA approval for Merck & Co., Inc.s MRK Keytruda as the second PD-1 inhibitor for the treatment of relapsed Hodgkin lymphoma in Mar 2017.

2017 Outlook Update

The company reiterated its outlook for net sales of Adectris to be in the range of $280 million to $300 million. Although the company expects royalty revenues to decrease in the second quarter, it continues to expect it in the range of $50 million to $55 million for the full year.

Pipeline Update

Seattle Genetics continues to work on expanding Adcetris label further through 3 phase III trials. We note that the company reported positive data from ALCANZA phase III study recently and is planning to submit a supplemental Biologics License Application (BLA) by mid-2017.

The company expects top-line data from the phase III ECHELON-1 study (frontline classical Hodgkin lymphoma) during 2017 while top-line data from the ECHELON-2 study (frontline CD30-expressing mature T-cell lymphoma) should be out in 2018 (previously expected in the 2017 to 2018 timeframe).

In addition, the company continues to enroll patients in the phase III study on vadastuximabtalirine in combination with hypomethylating agents in older patients with newly diagnosed acute myeloid leukemia. Also, the company plans to start a phase II study in younger patients with newly diagnosed AML in the second half of 2017.

Our Take

While the company missed bottom line expectations, sales came in ahead of estimates in the first quarter of 2017. With the approval of Keytruda, investors should focus on Adcetris sales in the upcoming quarters. However, the FDA has lifted the clinical hold on two phase I trials of its candidate, vadastuximabtalirine in acute myeloid leukemia (AML) in March.

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Seattle Genetics, Inc. Price, Consensus and EPS Surprise | Seattle Genetics, Inc. Quote

Zacks Rank & Stocks to Consider

Seattle Genetics carries a Zacks Rank #4 (Sell).

A couple of better-ranked stocks in the health care sector include Heska Corporation HSKA and Regeneron Pharmaceuticals, Inc. REGN. While Heska sports a Zacks Rank#1 (Strong Buy), Regeneron carries a Zacks Rank #2 (Buy). You can see the complete list of todays Zacks #1 Rank stocks here.

Heskas earnings estimates increased from $1.53 to $1.65 for 2017 and from $1.80 to $2.01 for 2018 over the last 60 days. The company posted positive earnings surprises in eachof the four trailing quarters with an average beat of 291.54%.

Regenerons earnings estimates remained almost stable for 2017 and 2018, respectively, over the past 30 days. The company recorded positive earnings surprises in three of the last four quarters, with the average being 8.08%.

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Seattle Genetics (SGEN) Q1 Loss Widens, Sales Top Estimates - Yahoo Finance

Genetics

Can’t hack it in high altitudes? It’s all about genetics – CBC.ca

Try to walk a mile in a Tibetan person's shoes and you will fail miserably.That's because they have evolved to live at high altitudes better than anyone else on the planet.

Just in time for the climbing season to open on Mount Everest, anew study from the University of Texas, Houston, tells us about the genes behind this incredible ability.

It's estimatedthat humans have been continually living on the Tibetan plateau for almost 60,000 years. That gives a lot of time for adaptations to develop and populations to evolve in order to live better in these extreme conditions.

When you are up there high on the Tibetan plateau, life is tough.When I was in Tibet in 2010 and again in 2011, it was amazing how hard it was just to walk around.There are a lotof high-altitude places in the world, but Lhasa, at an altitude of 3,600 metres, is one of the highest places where humans have lived for thousands of years.

I spoke with Chad Huff, the senior author of the study.

"Tibetan high-altitude adaptation is perhaps the most extreme example we have of human adaptation where most individuals from low altitude have a very difficult time at 15,00 feet," he said. "Tibetans survive and thrive there, so it's really fascinating as to the genetics behind those adaptations."

And that's what this new paper, published in the Open Access Journal PLOS, investigated; they looked at the genetic changes in Tibetans that make them such great mountaineers.

After sequencing the complete genomes of 27 Tibetans, they identified five possible genes that relate to the Tibetans' ability to thrive at such high altitudes.Two of them, EGL1andEPAS1,have been previously found,and both seem to be related to how much hemoglobin is in the blood.

Hemoglobin is what carries oxygen in the blood. When you acclimate to higher altitudes,your body adjusts to the low oxygen concentrations by making more hemoglobin to suck up more of that hard-to-get oxygen.

The two genes, EGL1andEPAS1,found in Tibetan populations seem to be doing the opposite: they seem to be decreasing the hemoglobin concentrations.

This makes no sense, until you realize that having blood thick with hemoglobin makes your heart pump harder and your vessels strain more. Having a heart that works harder all the time, coupled with vessels that are strained, doesn't add up to a healthy individual.This is one problem observed with the ethnic Han population, the dominant population of lowland Chinawho are trying to live in the Tibetan plateau to take advantage of some of the resource extraction that is happening.The non-Tibetansshow significant negative health effects, while the Tibetans show no such problems.

There's no exact number yet, but it looks like some of these genetic changes are as recent as 10,000 years old.The most common adaptation, and the one with the most variability in the population, is 45,000 years old. But still, that's pretty quick in evolutionary time, especially in humans who reproduce relatively slowly.

Chad Huff offers more insight.

"It is quite fast for humans. There are a few examples of somewhat faster evolution in humans, the lactase persistence in Europeans is one example; it's a few thousand years younger, butEPAS1andEGL1are among the strongest signals and fastest sweeps in human evolution."

You have to remember, the selective pressures the forces that pick the strongest individuals to survive are strong up there on the plateau. I can speak from experience.When I was hiking to Everest base camp it was one step breathe, two steps, breathe.I couldn't believe how hard it was,and there was my Tibetan guide, sprintingby, seemingly unfazed.

Researchers were interested in how Sherpa's work at such high altitudes. (Craig Steinback)

Yes, the study showed that Tibetans are literally genetically superior to anyone else trying to summit.

When I was there my guide had been to camp 4, the last one before the summit, more than a handful of times. That would be considered a huge success for most people attempting to climb Everest, and yetit was largely uncelebrated.

There is no doubt that in mountaineering, the earlier lack of credit for the Sherpas has been a huge controversy. Maybe because it is so much easier for them, it's almost as if the rest of the world hasn't been acknowledging the incredible risksthey take because it's so natural.

And yet, more Sherpas have been killed on Everest in the last three climbing seasons than ever before.And now we embark on another Everest climbing season, poised to the busiest ever.

The Sherpas are the lifeline to the mountain;no one can climb it without them.They set the routes, fix the ropes,carry a lot of the gearand do it better than anyone else on the planet.

Summitting Everest is an absolutely monumental achievement, and what the Tibetan people have survived and how they've adapted to the life in the mountains is equally monumental.

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Can't hack it in high altitudes? It's all about genetics - CBC.ca

Embryology

Embryology in action – York News-Times

YORK The 11 lively third-graders in Jillian Starks class received an eyes- and hands-on life lesson.

With help from York County Extension 4-H assistant Tanya Crawford and 4-H director Megan Burda, Stark and her students incubated, carefully monitored and hatched eggs in their classroom..

Embryology is a 4-H School Enrichment project taught in almost every elementary school across the state. The program is designed for grades 2-4. The Extension Office provides the incubator, eggs and educational materials.

In classroom discussions Crawford taught Starks students about eggs and the birds from tiny to very large - that lay them. They also found homes for the newly-hatched chicks.

Once Stark took up the Extension offer and signed on, Crawford came to class to introduce the project and then visited again with Burda to candle the eggs.

Students of all ages, Crawford said, enjoy taking care of eggs and anticipating the arrival of baby chicks.

We learn about the similarities and differences between chickens, as well as study the development of life. The program usually starts in March or April. I would love to bring embryology to your classroom, Crawford wrote in an earlier invitation to local teachers.

Embryology ran for 21 days during which time Stark and her students carefully checked temperatures in the hatching container and filled water to last over the weekends.

In the past, I have done up to three visits in the classroom (one per week), Crawford said, to deliver the following curriculum:

Lesson 1 introduce embryology and deliver eggs

Lesson 2 talk about egg development and candle the eggs

Lesson 3 talk about brooding and share egg collection

Article continued below advertisement

The eggs for Starks class came from Clare Quandt from Stromsburg and Jill and Ryan Koch from York.

For her part, Stark said, I decided to take on the embryology project because I thought it would be a great way for the students to learn about eggs and their connection to agriculture. The 4-H Extension office brought the supplies, including the incubator and the 24 eggs. After seven days the students candled the eggs to see if they were fertilized. After 14 days the students candled them again to check on their growth, she said.

By day 20 the eggs had begun to hatch.

The Extension Office then provided the necessary supplies to keep the chicks in the classroom for a few days, she explained.

The students have been really engaged throughout the whole process, she added. They were excited to watch the (chicks) hatch from the egg and hold them after the feathers had dried. Not only are they learning about science behind the chick, but they are also learning about the responsibility that comes along with having a pet. The experience has been great overall and I cant wait for next year.

Teachers of grades 2-4 are invited to contact Crawford about bringing the project to their classrooms, too.

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Embryology in action - York News-Times

Cell Biology

Researchers 3-D Bioprint Cartilage – Laboratory Equipment

A team of researchers at Sahlgrenska Academy has managed to generate cartilage tissue by printing stem cells using a 3D-bioprinter. The fact that the stem cells survived being printed in this manner is a success in itself. In addition, the research team was able to influence the cells to multiply and differentiate to form chondrocytes (cartilage cells) in the printed structure.

The findings have been published in Natures Scientific Reports magazine. The research project is being conducted in collaboration with a team of researchers at the Chalmers University of Technology who are experts in the 3D printing of biological materials. Orthopedic researchers from Kungsbacka are also involved in the research collaboration.

The team used cartilage cells harvested from patients who underwent knee surgery, and these cells were then manipulated in a laboratory, causing them to rejuvenate and revert into pluripotent stem cells, i.e. stem cells that have the potential to develop into many different types of cells. The stem cells were then expanded and encapsulated in a composition of nanofibrillated cellulose and printed into a structure using a 3D bioprinter. Following printing, the stem cells were treated with growth factors that caused them to differentiate correctly, so that they formed cartilage tissue.

The publicationis the result of three years of hard work.

In nature, the differentiation of stem cells into cartilage is a simple process, but its much more complicated to accomplish in a test tube. Were the first to succeed with it, and we did so without any animal testing whatsoever," says Stina Simonsson, Associate Professor of Cell Biology, who lead the research teams efforts.

Most of the teams efforts had to do with finding a procedure so that the cells survive printing, multiply and a protocol that works that causes the cells to differentiate to form cartilage.

"We investigated various methods and combined different growth factors. Each individual stem cell is encased in nanocellulose, which allows it to survive the process of being printed into a 3D structure. We also harvested mediums from other cells that contain the signals that stem cells use to communicate with each other so called conditioned medium. In laymans terms, our theory is that we managed to trick the cells into thinking that they arent alone, clarifies Simonsson. "Therefore,the cells multiplied before we differentiated them."

A key insight gained from the teams study is that it is necessary to use large amounts of live stem cells to form tissue in this manner.

The cartilage formed by the stem cells in the 3D bioprinted structure is extremely similar to human cartilage. Experienced surgeons who examined the artificial cartilage saw no difference when they compared the bioprinted tissue to real cartilage, and have stated that the material has properties similar to their patients natural cartilage. Just like normal cartilage, the lab-grown material contains Type II collagen , and under the microscope the cells appear to be perfectly formed, with structures similar to those observed in samples of human-harvested cartilage.

The study represents a giant step forward in the ability to generate new, endogenous cartilage tissue. In the not too distant future, it should be possible to use 3D bioprinting to generate cartilage based on a patients own, backed-up stem cells. This bioprinted tissue can be used to repair cartilage damage, or to treat osteoarthritis, in which joint cartilage degenerates and breaks down. The condition is very common one in four Swedes over the age of 45 suffer from some degree of osteoarthritis.

In theory, this research has created the opportunity to generate large amounts of cartilage, but one major issue must be resolved before the findings can be used in practice to benefit patients.

The structure of the cellulose we used might not be optimal for use in the human body. Before we begin to explore the possibility of incorporating the use of 3D bioprinted cartilage into the surgical treatment of patients, we need to find another material that can be broken down and absorbed by the body so that only the endogenous cartilage remains, the most important thing for use in a clinical setting is safety explains Simonsson.

Original post:
Researchers 3-D Bioprint Cartilage - Laboratory Equipment

Cell Biology

Kimmel Scholar Award propels immune cell cancer research – Davis Enterprise

UC Davis assistant professor Sean Collins of the department of microbiology and molecular genetics has received a prestigious, two-year $200,000 award that will help advance the use of immune cells for cancer therapies.

The Kimmel Scholar Award is given to 15 of the nations most promising young researchers leading the fight against cancer.

Collins seeks to understand how immune cells process information, make decisions and respond to threats in the human body. His research explores the basic molecular mechanisms that allow immune cells to navigate to infection sites.

When there is an injury or infection, our bodies respond by sending specialized immune cells to evaluate and intercept foreigner invaders. These immune cells also help defend the body against tumors. However, in order to fight infections or tumors, the cells must first find the right location in the body, and the path to get there can be complicated.

By identifying key principles and molecular pieces, we hope to reengineer these processes to direct immune cells to tumor locations, Collins said.

The hope is that by guiding the seek and destroy ability of these immune cells, in combination with other therapeutic strategies, they will be able to more effectively target and destroy tumors.

Ive spent most of my career so far focused on understanding basic mechanisms like how a cell processes information about its environment, Collins said, but this is a new direction to try and apply some of those findings in a direct, medically relevant way to help develop strategies for cancer therapy.

From sports statistics to cell biology Professor Wolf-Dietrich Heyer, chair of the department of microbiology and molecular genetics, recognizes Collins as a rising star with a bright future.

Professor Collins work is an elegant combination of cutting-edge cell biology paired with rigorous quantitative analysis and creative mathematical modeling, Heyer said. His focus on immune cells will provide the underpinning for novel approaches in harnessing the bodys immune system in anti-cancer therapy.

Collins interest in science grew from a childhood fascination with solving problems.

At some level, it started with an interest in computer programming and statistics in sports, he said.

As a child, Collins and his twin brother were big sports statistics buffs, and would play sports simulation games on the computer. One day, their game malfunctioned, displaying a cryptic error message. So the two brothers spent the next few months figuring out how the game worked and eventually fixed it.

The Kimmel Scholar Award is sponsored by the Sidney Kimmel Foundation, which has funded more than 260 cancer researchers since its founding in 1993. Collins is the second researcher from UCD to be honored with this award, after professor Ken Kaplan of the department of molecular and cellular biology in 2001.

UC Davis News

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Kimmel Scholar Award propels immune cell cancer research - Davis Enterprise

Anatomy

Grey’s Anatomy recap: ‘Don’t Stop Me Now’ – EW.com

One patient had worms breeding inside her. The end.

Just kidding. There was more to this episode, but honestly, the pasta-like worms were everywhere even in scenes most definitelynotabout the worms, you were thinking about the worms. Or if, like me, you are getting over a stomach bug, you were thinking, Please dont throw up, please dont throw up, please dont throw up. Also? Spaghetti is ruined. Forever.

All you really need to know about the worms is that a patient had them, her new boyfriend ditched her once he found out, and then he returned later (after the worms were cleared from his girlfriend, it should be noted), once he realized he was being a butthead. Oh, and April got one in her hair. And you thought licewere bad!

In non-worm news, Bailey notices Richard and Catherine arent speaking, and she wants to fix it because of course she does. She spends the entire episode trying to figure out how to get them back on speaking terms. April eventually clues Bailey in on something: She and Catherine are very, very alike. Soon after, Baileys confronting her sort-of twin about how she needs to woman up and talk to her husband. Shes pretty convincing, too, both because shes Bailey, and Bailey could probably convince you breeding worms in your intestines is a good idea (sorry), and because she knows a thing or two about marriage shes been through two, after all.

So she does her Bailey thing, telling Catherine things like, You dont know how to be married, and, in the episodes best moment, I plan to grow old with my man and have old people sex. Dont you want to have old people sex, Catherine? she asks. She tells Catherine to set her ego aside and talk to her husband, make up with him. Very quickly we find out that Catherinedoeswant to have old people sex, because shes visiting Richard and joyfully looking at his pictures of the worms. At least something good came out of them?

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Grey's Anatomy recap: 'Don't Stop Me Now' - EW.com

Anatomy

‘Grey’s Anatomy’ Recap: Maggie Finds Out About Meredith and Riggs – Us Weekly

Sisters before misters! Meredith Grey (Ellen Pompeo) might end her latest fling to make things right with Maggie Pierce (Kelly McCreary) on Greys Anatomy. Also, a few couples reconnected during the Thursday, April 27, episode, so love was definitely in the air at Grey Sloan Memorial Hospital. Check out the five biggest moments from the episode!

Meredith and Maggie Drama

Meredith decided it was finally time for her to tell Pierce about her relationship with Nathan Riggs (Martin Henderson). However, before she got a chance, Pierce saw the two flirting during a press conference and figured it out. She gave Meredith the cold shoulder all day.

This isnt about Nathan. Its you. I trusted you to be honest with me, Pierce said. I thought I could count on you. Mer, dont let your girl down. You have to fix this.

Matchmaker in Heaven

Miranda Bailey (Chandra Wilson) was obsessed with fixing Richard Webbers (James Pickens Jr.) marriage. Her husband, Ben Warren (Jason Winston George), told her to stay out of it, but she couldnt help herself and enlisted April Kepner (Sarah Drew) for help.

Just because you feel responsible doesnt mean you have to fix it. Stay out of it, Catherine Avery (Debbie Allen) told Bailey.

I know it can feel impossible at times, but you can figure it out, Bailey said.

A Short Reconnection

After Amelia Shepherd (Caterina Scorsone) performed a heart-wrenching surgery, she could not keep it together. She stayed cool in front of the patients, but once it all was over, she broke down in an elevator. Owen Hunt (Kevin McKidd) got on the elevator and saw her, and the two stood there hugging in silence as Amelia sobbed. When Hunt got to his floor, he got off without saying a word. Although it wasnt much, maybe these two can get back on track.

Working With Worms

Webber had a busy day extracting worms from a patients body. It seemed like Bailey actually got through to Catherine, and Webbers wife visited him as he was getting ready to sleep in the hospital.

I was wrong, she said. The two didnt really discuss their problems, but at least they bonded over the photos Webber took of the worm surgery. They are totally meant for one another.

Breaking Up?

Meredith came home to Maggie sitting on the couch and told her they were going out. Maggie didnt want to, but Mer wouldnt take no for an answer.

I am very sorry that I screwed up and I hurt you, Meredith said. But youre my sister and Im sure as hell not losing you over a guy.

That must have been exactly what Pierce wanted to hear, because she agreed to go. Hopefully, this will finally be the end of the Meredith-Riggs rollercoaster. They keep trying to be a thing, but it just doesnt work. Plus, Meredith and Pierce are sisters. They cant ruin that over a guy.

Tell Us: Do you think Meredith will finally break up with Riggs?

Greys Anatomy airs Thursdays at 8 p.m. on ABC.

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'Grey's Anatomy' Recap: Maggie Finds Out About Meredith and Riggs - Us Weekly

Anatomy

‘Big Bang Theory,’ ‘Grey’s Anatomy’ and ‘Masterchef Junior’ adjust up; ‘The Catch’ adjusts down: Thursday final ratings – TVbytheNumbers

Final broadcast primetime live + same-day ratings for Thursday, April 27, 2017

The Big Bang Theory avoided a new series low among adults 18-49 and finished ahead of its last episode by adjusting up from Thursdays early ratings to the finals. The shows 18-49 rating came up two tenths of a point to 2.6, 0.1 higher than its previous series low two weeks ago.

Greys Anatomy (1.8) and Masterchef Junior (0.8) also adjusted up. The Catch came down a tenth to 0.6.

Upward adjustments in adults 18-49 are in blue; downward adjustments are in red.

Network averages:

Definitions: Rating: Estimated percentage of the universe of TV households (or other specified group) tuned to a program in the average minute. Ratings are expressed as a percent. Fast Affiliate Ratings: These first national ratings are available at approximately 11 a.m. ETthe day after telecast. The figures may include stations that did not air the entire network feed, as well as local news breaks or cutaways for local coverage or other programming. Fast Affiliate ratings are not as useful for live programs and are likely to differ significantly from the final results, because the data reflect normal broadcast feed patterns. Share (of Audience):The percent of households (or persons) using television who are tuned to a specific program, station or network in a specific area at a specific time. Time Shifted Viewing:Program ratings for national sources are produced in three streams of data Live, Live +Same-Day and Live +7 Day. Time-shifted figures account for incremental viewing that takes place with DVRs. Live+SD includes viewing during the same broadcast day as the original telecast, with a cut-off of 3 a.m. local time when meters transmit daily viewing to Nielsen for processing. Live +7 ratings include viewing that takes place during the 7 days following a telecast.

Source: The Nielsen Company.

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'Big Bang Theory,' 'Grey's Anatomy' and 'Masterchef Junior' adjust up; 'The Catch' adjusts down: Thursday final ratings - TVbytheNumbers