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Conservative and Liberal Brains Might Have Some Real Differences – Scientific American

In 1968 a debate was held between conservative thinker William F. Buckley, Jr., and liberal writer Gore Vidal. It was hoped that these two members of opposing intellectual elites would show Americans living through tumultuous times that political disagreements could be civilized. That idea did not last for long. Instead Buckley and Vidal descended rapidly into name-calling. Afterward, they sued each other for defamation.

The story of the 1968 debate opens a well-regarded 2013 book called Predisposed, which introduced the general public to the field of political neuroscience. The authors, a trio of political scientists at the University of Nebraska-Lincoln and Rice University, argued that if the differences between liberals and conservatives seem profound and even unbridgeable, it is because they are rooted in personality characteristics and biological predispositions.

On the whole, the research shows, conservatives desire security, predictability and authority more than liberals do, and liberals are more comfortable with novelty, nuance and complexity. If you had put Buckley and Vidal in a magnetic resonance imaging machine and presented them with identical images, you would likely have seen differences in their brain, especially in the areas that process social and emotional information. The volume of gray matter, or neural cell bodies, making up the anterior cingulate cortex, an area that helps detect errors and resolve conflicts, tends to be larger in liberals. And the amygdala, which is important for regulating emotions and evaluating threats, is larger in conservatives.

While these findings are remarkably consistent, they are probabilities, not certaintiesmeaning there is plenty of individual variability. The political landscape includes lefties who own guns, right-wingers who drive Priuses and everything in between. There is also an unresolved chicken-and-egg problem: Do brains start out processing the world differently or do they become increasingly different as our politics evolve? Furthermore, it is still not entirely clear how useful it is to know that a Republicans brain lights up over X while a Democrats responds to Y.

So what can the study of neural activity suggest about political behavior? The still emerging field of political neuroscience has begun to move beyond describing basic structural and functional brain differences between people of different ideological persuasionsgauging who has the biggest amygdalato more nuanced investigations of how certain cognitive processes underlie our political thinking and decision-making. Partisanship does not just affect our vote; it influences our memory, reasoning and even our perception of truth. Knowing this will not magically bring us all together, but researchers hope that continuing to understand the way partisanship influences our brain might at least allow us to counter its worst effects: the divisiveness that can tear apart the shared values required to retain a sense of national unity.

Social scientists who observe behaviors in the political sphere can gain substantial insight into the hazards of errant partisanship. Political neuroscience, however, attempts to deepen these observations by supplying evidence that a belief or bias manifests as a measure of brain volume or activitydemonstrating that an attitude, conviction or misconception is, in fact, genuine. Brain structure and function provide more objective measures than many types of survey responses, says political neuroscientist Hannah Nam of Stony Brook University. Participants may be induced to be more honest when they think that scientists have a window into their brains. That is not to say that political neuroscience can be used as a tool to read minds, but it can pick up discrepancies between stated positions and underlying cognitive processes.

Brain scans are also unlikely to be used as a biomarker for specific political results because the relationships between the brain and politics is not one-to-one. Yet neurobiological features could be used as a predictor of political outcomesjust not in a deterministic way, Nam says.

To study how we process political information in a 2017 paper, political psychologist Ingrid Haas of the University of Nebraska-Lincoln and her colleagues created hypothetical candidates from both major parties and assigned each candidate a set of policy statements on issues such as school prayer, Medicare and defense spending. Most statements were what you would expect: Republicans, for instance, usually favor increasing defense spending, and Democrats generally support expanding Medicare. But some statements were surprising, such as a conservative expressing a pro-choice position or a liberal arguing for invading Iran.

Haas put 58 people with diverse political views in a brain scanner. On each trial, participants were asked whether it was good or bad that a candidate held a position on a particular issue and not whether they personally agreed or disagreed with it. Framing the task that way allowed the researchers to look at neural processing as a function of whether the information was expected or unexpectedwhat they termed congruent or incongruent. They also considered participants own party identification and whether there was a relationship between ideological differences and how the subjects did the task.

Liberals proved more attentive to incongruent information, especially for Democratic candidates. When they encountered such a position, it took them longer to make a decision about whether it was good or bad. They were likely to show activation for incongruent information in two brain regions: the insula and anterior cingulate cortex, which are involved in helping people form and think about their attitudes, Haas says. How do out-of-the-ordinary positions affect later voting? Haas suspects that engaging more with such information might make voters more likely to punish candidates for it later. But she acknowledges that they may instead exercise a particular form of bias called motivated reasoning to downplay the incongruity.

Motivated reasoning, in which people work hard to justify their opinions or decisions, even in the face of conflicting evidence, has been a popular topic in political neuroscience because there is a lot of it going around. While partisanship plays a role, motivated reasoning goes deeper than that. Just as most of us like to think we are good-hearted human beings, people generally prefer to believe that the society they live in is desirable, fair and legitimate. Even if society isnt perfect, and there are things to be criticized about it, there is a preference to think that you live in a good society, Nam says. When that preference is particularly strong, she adds, that can lead to things like simply rationalizing or accepting long-standing inequalities or injustices. Psychologists call the cognitive process that lets us do so system justification.

Nam and her colleagues set out to understand which brain areas govern the affective processes that underlie system justification. They found that the volume of gray matter in the amygdala is linked to the tendency to perceive the social system as legitimate and desirable. Their interpretation is that this preference to system justify is related to these basic neurobiological predispositions to be alert to potential threats in your environment, Nam says.

After the original study, Nams team followed a subset of the participants for three years and found that their brain structure predicted the likelihood of whether they participated in political protests during that time. Larger amygdala volume is associated with a lower likelihood of participating in political protests, Nam says. That makes sense in so far as political protest is a behavior that says, Weve got to change the system.

Understanding the influence of partisanship on identity, even down to the level of neurons, helps to explain why people place party loyalty over policy, and even over truth, argued psychologists Jay Van Bavel and Andrea Pereira, both then at New York University, in Trends in Cognitive Sciences in 2018. In short, we derive our identities from both our individual characteristics, such as being a parent, and our group memberships, such as being a New Yorker or an American. These affiliations serve multiple social goals: they feed our need to belong and desire for closure and predictability, and they endorse our moral values. And our brain represents them much as it does other forms of social identity.

Among other things, partisan identity clouds memory. In a 2013 study, liberals were more likely to misremember George W. Bush remaining on vacation in the aftermath of Hurricane Katrina, and conservatives were more likely to falsely recall seeing Barack Obama shaking hands with the president of Iran. Partisan identity also shapes our perceptions. When they were shown a video of a political protest in a 2012 study, liberals and conservatives were more or less likely to favor calling police depending on their interpretation of the protests goal. If the objective was liberal (opposing the military barring openly gay people from service), the conservatives were more likely to want the cops. The opposite was true when participants thought it was a conservative protest (opposing an abortion clinic). The more strongly we identify with a party, the more likely we are to double down on our support for it. That tendency is exacerbated by rampant political misinformation and, too often, identity wins out over accuracy.

If we understand what is at work cognitively, we might be able to intervene and try to ease some of the negative effects of partisanship. The tension between accuracy and identity probably involves a brain region called the orbitofrontal cortex, which computes the value of goals and beliefs and is strongly connected to memory, executive function and attention. If identity helps determine the value of different beliefs, it can also distort them, Van Bavel says. Appreciating that political affiliation fulfills an evolutionary need to belong suggests we should create alternative means of belongingdepoliticizing the novel coronavirus by calling on us to come together as Americans, for instance. And incentivizing the need to be accurate could increase the importance accorded that goal: paying money for accurate responses or holding people accountable for incorrect ones have been shown to be effective.

It will be nearly impossible to lessen the partisan influences before the November 3 election because the volume of political information will only increase, reminding us of our political identities daily. But here is some good news: a large 2020 study at Harvard University found that participants consistently overestimated the level of out-group negativity toward their in-group. In other words, the other side may not dislike us quite so much as we think. Inaccurate information heightened the negative bias, and (more good news) correcting inaccurate information significantly reduced it.

The biology and neuroscience of politics might be useful in terms of what is effective at getting through to people, Van Bavel says. Maybe the way to interact with someone who disagrees with me politically is not to try to persuade them on the deep issue, because I might never get there. Its more to try to understand where theyre coming from and shatter their stereotypes.

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Conservative and Liberal Brains Might Have Some Real Differences - Scientific American

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Yale finds neuron behind fatal anorexia, and solution in high-fat diet – Yale News

Researchers have long known that many people (mostly women) suffering from anorexia face a high risk of death. But, until now, they didnt know what causes the eating disorder to turn fatal.

In a research letter published in the Oct. 26 edition of Nature Metabolism, Yale researchers describe a specific neuron that appears to play an important role in whether anorexia becomes deadly.

They also discovered a potential treatment: a high-fat diet.

Over the last 25 years, our work has focused on understanding what drives hunger, said author Tamas Horvath, the Jean and David W. Wallace Professor of Comparative Medicine and professor of neuroscience and of obstetrics, gynecology, and reproductive sciences. We wondered if neurons in the brain which are working at a high level when someone is dieting could be participating in some aspect of the disease.

For the study, the researchers looked at a specific neuron that is active during food restriction, called the hypothalamic agouti-related peptide (AgRP), in food-restricted, exercising mice. They found a direct relationship between the workings of the neuron and the animals likelihood of dying. In fact, all animals on a food-restricted, high-exercise diet whose AgRP neurons were inhibited died within 72 hours.

If we diminished these neurons in animals who ate little and exercised compulsively, they died, said Horvath, who is also chair of the Department of Comparative Medicine and director of the Yale Program in Integrative Cell Signaling and Neurobiology of Metabolism.

Lowering levels of these neurons proved fatal, Horvath said, because they are needed to help the body access alternative forms of fuel namely fat in the absence of eating, combined with intense exercise. If these neurons dont function, you are not able to mobilize fuels from fat stores, he said.

But when they provided fatty food to the mice with decreased AgRP activity they found that death [was] completely prevented. This finding could suggest a new tactic for treating anorexia in people, Horvath said. If you are a person dying from anorexia and eat foods containing elevated fat, you may survive, he said.

Anorexia nervosa is an eating disorder that affects mainly adolescent girls. Those with the disorder severely restrict their eating, fear gaining weight, and exercise compulsively. Some 20 million women suffer from anorexia, which has the highest mortality rate of any mental illness.

Horvath and other Yale researchers are now extending their research to identify which fats may work best in preventing anorexia from becoming lethal. Many people with this disorder are in the care of medical professionals, and theres an opportunity to bring these findings to the human population, he said.

Other Yale researchers who contributed to the research letter include first author Maria Miletta, postdoctoral associate in comparative medicine; Onur Iyilikci, postdoctoral associate in comparative medicine; Marya Shanabrough, research associate in comparative medicine; Matija Sestan-Pesa, postdoctoral associate in comparative medicine; Caroline Zeiss, professor of comparative medicine; and Marcelo Dietrich, associate professor of comparative medicine and of neuroscience. The work was supported by the Klarman Family Foundation and the National Institutes of Health.

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The Effects of Sugar on the Brain How Sugar Affects the Brain – GoodHousekeeping.com

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Sugar rush. Sugar high. Sugar buzz. Sugar slump.

We've all heard these phrases that describe what our brains feel like when we've had too much of a sweet treat. But heres the thing about sugar and your brain: Your noggin needs it because sugar is its main fuel. Our brains run on glucoseit fuels our cells, including our brain cells. Were evolutionarily programmed to like sweets since theyre a great energy source. And when glucose levels are low in the blood (say, if you have hypoglycemia, or sometimes if it's just been a while since you've had something to eat), brain fog sets in, you fumble for words, you forget where you put down your phone, you look for a place to curl up for a quick nap.

When theres too much sugar in the system, however, that can set off some really bad stuff in your body and your brain.

When sugar hits our tongue, it activates certain taste buds that send a signal up to the brain, including the cerebral cortex, says Nicole Avena, Ph.D., assistant professor of neuroscience at Mount Sinai School of Medicine, who wrote a book about sugar addiction. The signal activates the brains reward system; dopamine (a feel-good brain chemical) is released and the behavior is reinforced, which makes us want to repeat it (mmmm, thats nice, give me more!).

Sugar is rare among dopamine-producing foods, Avena points out. Most of the time when we eat something new and tasty, dopamine is released the first time we taste it, she explains. This is an evolutionary advantage to help us to pay attention to new and different tastes, in case they make us sick. If we eat something new and don't get sick, typically the dopamine response then goes away the next timeso basically we only release dopamine in response to eating new foods. However, sugar is different. Its more like what happens with a drug of abuse, where dopamine is released every single time its consumed. Eating lots of sugar will continue to feel rewarding because the dopamine level doesnt balance out, which it does when eating healthier foods. So sugar does act a bit like a drug in our system which is why people get hooked on sugary foods.

We used to have to forage for sweet foodsbut no more, of course. Our world is filled with sugary foods and not just the obvious donuts and super-sweetened, super-large latte drinks. A host of surprising foods from ketchup to salad dressing to marinara sauce have sugar added to them. But our brains still function like sugar is a rarity.

When you repeatedly activate that reward system, your brain adapts and actually rewires itself, and you crave more and more. And research on rats turned up this finding: When they were fed high-sugar diets, the rats brains released less of a certain chemical that helps bodies put on the brakes, so they were less able to stop eating it.

Those sugar-saturated rats also werent as good at certain memory tasks, which showed that certain brain functions in the prefrontal area and hippocampus (an important area for memory formation and retention) were impaired. Other rat studies showed that a high sugar diet increased inflammation in this area, which also affected short term memory.

You may not be aware that theres a strong connection between our gut and our brainand sugar comes into play here as well. When that sugary thing youve eaten hits your gut, Avena says, it activates sugar receptors there too, which signal the brain to release insulin to deal with the extra sugar youve eaten. To explain further: Excess sugar drives the pancreas to produce extra insulin, a hormone involved in blood sugar regulation. The insulin signals fat cells to store excessive amounts of glucose, fatty acids, and other calorie-rich substances. As a result, too few calories remain in the bloodstream, so the brain thinks its now low on fuel (since it has those very high energy needs). So your hunger level rises quickly. And sugar is appealing then because it provides quick energy. Thus, the cycle begins again. And thus, cravings for more and more brownies or ice cream or candy.

Sugar Shock: The Hidden Sugar in Your Food and 100+ Smart Swaps to Cut Back

Want to get your brain in line with a healthier sugar level? Your brain can readapt when you cut back on sugar, and you wont crave it as much, says Avena. However, it can take awhile, even months, for this to happen, depending on the severity of dependence on sugar that one has.

To find out more about how to start cutting back on sugar and shake off a sugar dependence, try a 7-day detox plan. When you can successfully cut back, there's a payoff, besides a much healthier body: Just a small taste of, say, that delicious chocolate brownie will be enough of a sweet treat to satisfy you.

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How a worm may yield insights into the gut-brain relationship – MIT News

The naked eye can barely spot the transparent nematodes at the center of PhD student Gurrein Madans neuroscience research. While C. elegans worms may initially seem an unassuming test subject for a graduate student who investigates the intricacies of gut-brain signaling, many of the genes found in C. elegans have counterparts in the human brain. Gurreins research could yield new insights into the gut-brain relationship, which may have practical health implications for humans.

Gurrein works in the lab of Steven Flavell, the Lister Brothers Career Development Assistant Professor in the Picower Institute for Learning and Memory and Department of Brain and Cognitive Sciences at MIT. There, researchers address some of neurosciences most essential questions, using C. elegans as a model. The lab centers around understanding how neuromodulatory systems such as those cellular systems that release, and are stimulated by, serotonin affect animal behavior. The millimeter-long C. elegans are an ideal model for this work because their nervous system, with just 302 neurons, has been well-characterized: It is the only animal on the planet where there is a full blueprint of how all of its brain cells are wired together, says Flavell. Combined with cutting-edge genetic and neural imaging technologies, the nematode model affords mechanistic studies of behavior from the scale of molecules to the whole brain.

Gurrein was recently named one of this years School of Science MathWorks Fellows. The fellowship is a one-year renewable opportunity for graduate students in the School of Science who use the software MATLAB to make impressive strides in their research. Funding for the fellowship is provided with support from MathWorks, founded by its president, John N. Little 78. MATLAB is used extensively by faculty, students, and researchers across the world and MITs campus to develop algorithms, computations, and simulations.

Gurreins project specifically looks at the neurons that line the C. elegans gut. These enteric neurons detect food and respond to changes in the animals nutritional state while receiving feedback from other parts of the brain. Gurrein studies the class of neurons that release serotonin, which has a profound influence on the animals feeding behavior.

Currently, we are investigating what receptors expressed in these enteric neurons regulate the neurons response to food, as well as to feedback from the rest of the nervous system, Gurrein says. By using genetics and neural imaging techniques, we attempt to uncover new molecular players involved in gut-brain signaling.

Dysregulation of gut-brain signaling has been linked to psychiatric disorders, such as depression and autism spectrum disorder, in humans. Diverse molecules, including neurotransmitters and inflammatory molecules, mediate the two-way communication between the gut and the brain. However, the specific pathways behind this relationship are not well understood. Gurrein hopes to uncover more about the signaling mechanisms driving the connection.

Much of our understanding of the fundamental pathways that control animal development and function comes from studies that originated in C. elegans, where basic genetic pathways were rapidly discovered, says Flavell. Lo and behold, in humans, the same pathways control the same cellular processes. Many of these pathways have then become targets for drug development to treat human disease.

Using MATLAB at nearly every step of her research from data collection and processing to analysis Gurrein was an excellent candidate for the MathWorks fellowship. I was excited to apply for two reasons. First, the fellowship was open to international students. Typically, international students are ineligible to apply to most fellowships out there. Second, MATLAB serves as the critical platform for comprehensively handling my data, Gurrein says.

Gurrein grew up in Amritsar in northwest India. Early in high school, Gurrein was placed in the sciences track, and upon graduation traveled to the United States for her undergraduate degree. During her sophomore year at Swarthmore College, she began research in a neurobiology lab and quickly realized how much she enjoyed the process of conducting scientific research. Moreover, she found the interdisciplinary nature of the neuroscience field exciting. After graduating with a BA in neuroscience in 2017, she immediately began her PhD at MIT.

I really like the innovative aspect of a PhD, Gurrein says. We are trained to expand the limits of what is known in our fields by being persistent, constantly troubleshooting, and coming up with new approaches to probe a question. I was initially considering medical school, but my research experiences led me to think that a PhD was probably a better fit for me.

Gurreins colleagues noticed her enthusiasm for scientific discovery immediately. Flavell says she impressed him right out of the gate. Within her first six months in the lab, Gurrein was instrumental in designing new experiments, conducting behavioral assays, and making notable discoveries that made their way into publications.

Gurrein has an enormous amount of drive and energy, always trying her best to make impactful discoveries, says Flavell. We have meetings once a week, and she always brings a critical eye to her own work, thinking about her datasets, what they mean, and how they give rise to new research directions. She wants to make sure the data she gets is really convincing and setting her on a path to making a true discovery.

The general topic of how the gut is influencing the brain is a relatively new field, Gurrein says. I think there is a lot of space for novel, exciting contributions.

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How a worm may yield insights into the gut-brain relationship - MIT News

Neuroscience Market Set to Witness Steady Growth through 2019-2029 – The Think Curiouser

Neuroscience Market size will reach xx million US$ by 2029, from xx million US$ in 2018, at a CAGR of xx% during the forecast period. In this study, 2018 has been considered as the base year and 2019-2029 as the forecast period to estimate the market size for Neuroscience.

This industry study presents the Neuroscience Market size, historical breakdown data 2014-2019 and forecast 2019-2029. The Private Plane production, revenue and market share by manufacturers, key regions and type; The consumption of Neuroscience Market in volume terms are also provided for major countries (or regions), and for each application and product at the global level.

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The Neuroscience Market has been reporting substantial growth rates with considerable CAGR for the last couple of decades. According to the report, the market is expected to grow more vigorously during the forecast period and it can also influence the global economic structure with a higher revenue share. The market also holds the potential to impact its peers and parent market as the growth rate of the market is being accelerated by increasing disposable incomes, growing product demand, changing consumption technologies, innovative products, and raw material affluence.

The study objectives are NeuroscienceMarket Report:

In this study, the years considered to estimate the market size of NeuroscienceMarket:

History Year: 2014 2018

Base Year: 2018

Estimated Year: 2019

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For the data information by region, company, type and application, 2018 is considered as the base year. Whenever data information was unavailable for the base year, the prior year has been considered.

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Aural Analytics, Inc. Granted Foundational U.S. Patent for Using Speech to Detect, Diagnose and Track Neurological Disease – Business Wire

SCOTTSDALE, Ariz.--(BUSINESS WIRE)--Aural Analytics, Inc., an industry leading speech neuroscience and speech analytics technology company, announced today that the United States Patent and Trademark Office issued US Patent number 10,796,715, an important foundational patent that broadly covers the use of a patients speech to diagnose and track neurological conditions including Parkinsons disease, amyotrophic lateral sclerosis (ALS), Huntingtons disease, multiple sclerosis (MS), traumatic brain injury (TBI), stroke and in other conditions where motor speech changes may occur.

Many neurological disorders that impact motor abilities result in changes to speech. The pattern of speech symptoms provides localizing information about where in the nervous system the disturbance is occurring. As these changes can be subtle and pre-clinical, objective algorithms for analyzing speech are required to detect them. This broad patent covers all algorithms and technology for extracting these speech characteristics, both in-the-clinic and remotely, as sensitive indicators to detect disease onset and track disease progression or treatment efficacy. Importantly, the patent protects the use of traditional clinical outcomes for the development of speech-based algorithms for diagnosis and tracking. This ensures Aural Analytics freedom to use current clinical gold standards as part of their computational analysis and development, while limiting the ability for others in the field to do the same.

The patent lays a foundation for the further integration of speech and voice analytics and biomarkers across the continuum of care in neurology. More frequent collection and objective assessments of speech could enable early intervention, better treatments and diagnostics, and can lead to personalized care pathways thereby improving patient outcomes and reducing long-term healthcare costs.

Our researchers and technologists have worked for decades building new inventions and breakthroughs in the field of speech analytics for health applications. This newly issued patent further strengthens our companys market leadership position and is a great step toward realizing our mission of using speech and voice to diagnose and track neurological and respiratory disease globally, across the continuum of care and throughout the lifespan, said Daniel Jones, CEO and co-founder of Aural Analytics.

The systems and methods covered by the patent are implemented into several speech elicitation and analysis modules in the industry leading A2E technology suite, an integration ecosystem that enables the ubiquitous collection and analysis of speech in clinical and research settings. A2E is currently available for integration into iOS, Android and web-based applications, in 14 languages, and specialized for use in neurology and respiratory disease. The patent will also be used in Aural Analytics award-winning clinical trials application, Speech Vitals.

About Aural Analytics, Inc.

Aural Analytics is the industrys leading speech neuroscience company building the worlds most advanced clinical-grade speech analytics platform for health applications across the lifespan. Currently available in 14 languages, Aural Analytics technologies provide rich, interpretable and validated metrics reflecting the neurological and respiratory health of its users. The company is founded on nearly three decades of NIH and NSF-funded research in speech neuroscience, is backed by dozens of high-caliber scientific publications, and has won several awards for its work in the field, including the prestigious Global SCRIP Award for Best Technology Development in Clinical Trials. Aural Analytics is based in Scottsdale, AZ. For more information, please visit auralanalytics.com or follow Aural Analytics on Twitter, LinkedIn, Medium and Facebook.

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Aural Analytics, Inc. Granted Foundational U.S. Patent for Using Speech to Detect, Diagnose and Track Neurological Disease - Business Wire

Team DIBS Incubator award to fund research on spinal cord injury recovery – Duke Department of Neurology

An interdisciplinary team from the Duke Departments of Neurology, Orthopedic Surgery, and Neurosurgery has received the Duke Institute for Brain Sciences (DIBS) $75,000 2020 Research Incubator Award to improve recovery after spinal cord injuries. These awards fund high-risk/high-return collaborative neuroscience research that crosses disciplinary boundaries, and is likely to draw external funding.

The Department of Orthopaedic Surgerys Timothy Faw, PhD, DPT (upper left) will lead the project, along with Neurologys Laskowitz, MD, MHS (upper right), and Haichen Wang, MD (lower left), and the Department of Neurosurgerys Muhammad Abd-El-Barr, MD, PhD (lower right).

Spinal cord injury can be a devastating and life changing event, and at present there are no effective neuroprotective therapies to improve outcomes. This funding will establish a multidisciplinary group of clinicians and researchers to help address this compelling unmet clinical need, said Laskowitz. The CN-105 peptide we are using would represent a first-in-class therapeutic, and and Dr. Faws team has already demonstrated improvement in clinically relevant animal models of spinal cord injury.

The collaborative project, A Novel Apolipoprotein E (apoE)-mimetic Pentapeptide to Improve Recovery in Acute Spinal Cord Injury, will attempt to reduce the bodys harmful early inflammatory response after a spinal cord injury. The team has developed small peptides that mimic the function of apolipoprotein E (apoE), which plays a critical role in mediating this neuroinflammation. This peptide, CN-105, has shown potential in clinical trials and has been shown to effectively cross the blood-brain barrier.

DIBS awarded five Incubator awards and one related Germinator award this year. Other funded projects will examine the relationship between tobacco use and chronic pain, how changes in the gut are communicated to the brain, the use of novel technologies to understand the neural mechanisms of Parkinsons disease, and the effects of toxins on the developing brain. Each project represents multiple departments and schools, including the Duke School of Medicine, the Pratt School of Engineering, Trinity College of Arts & Sciences, and the Nicholas School of the Environment.

We are pleased to be able to make these awards and highlight the value of interdisciplinary research, said DIBS Director Geraldine Dawson, PhD, in announcing the award recipients. Even during these financially challenging times, Dawson noted, we remain strongly committed to supporting collaboration and innovation in the neurosciences at Duke. We were especially pleased to see the breadth of departments and schools that received funding. Read more about the DIBS Incubator Awards here.

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Team DIBS Incubator award to fund research on spinal cord injury recovery - Duke Department of Neurology

Neuroscience Antibodies and Assays Market Business Opportunity, Segmentation, Industry Overview and Forecast Till 2023 – The Think Curiouser

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Neuroscience Antibodies and Assays Market Business Opportunity, Segmentation, Industry Overview and Forecast Till 2023 - The Think Curiouser