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Anatomy

Anatomy of a zipping up – Banners On The Parkway

Depending on who you ask, Xaviers win probability bottomed out at or slightly after the final media timeout. NumberFire puts the Musketeers down to their last 2.92% when Parker Jackson-Cartwright jarred the second of the two free throws he earned right before the break. KenPom doesnt label each play, but he had Xavier down to right around 3% when Kadeem Allen made a layup with 2:52 to play.

When Allens layup went through the strings, Xavier was down to its last breath in a shootout that had seen Arizona not be able to pull away despite averaging 1.29 points per possession in their 55 trips down the floor to that point. To get back on top before time ran out, Xavier would need to outscore Arizona by at least 8 in the remaining 2:52. With the tenor of the game to that point, scoring 8 was always going to be the easier part. Then the defense went to work.

Xavier was in a 2-3/1-1-3 here with JP and Q chasing all around the perimeter. That obviously left some gaps when the ball rotated quickly, and with 10 seconds left on the shot clock, Trier tried a long three that would have been a dagger. Instead it rimmed out, and JP dropped in from the wing to rake it out and go.

Back into the zone again. One of the few times I agreed with Chris Webbers analysis of the game was right here. Ten seconds into the possession, Trier dribbled into a 17-footer from the right elbow area for reasons best known only to himself. It was a hard brick, and a the ball caught a couple of hands before going out of bounds to Xavier.

Things really started to tighten up here for Arizona. The freedom with which they had been playing for most of the game was all but gone as it became clear momentum was on Xaviers side. After a lot of dribbling around, Trier got himself caught in a blind alley on the left side of the lane. Trier gave up the ball - which was smart - but he did so to Tre, which was of debatable strategic value to Arizona.

Maybe my favorite possession of the game. With the season in the balance, Xavier was back in a man look and Arizona tried to iso Ristic on the block against Tyrique Jones. Ristic banged hard twice against Jones to drive him under the bucket; he gained exactly no ground. Stuck ten feet out along the right baseline, he turned and tried a half hook shot from a really narrow angle. It never really had a chance. Twenty hands battled for the rebound, but a monitor review showed that the last one it touched belonged to a Wildcat.

A break here to stop talking about defense for a minute. While the refs looked at the monitor, Coach Dale scanned the Hickory High huddle and decided to run Jimmy Chitwood off of a screen as a decoy Coach Mack called Corner Rip High-Low Counter. This wasnt a one-off play that Mack drew up in the huddle. It wasnt - as Chris Webber suggested on the telecast - just a case of Sean OMara happening to seal his man after Xavier called Trevon Bluietts number. It was an action Xavier has run over and over in the Mack Era, one that any coach could find in some back issue of the newsletter that the Xavier basketball program produces. It was also the perfect call to put Xavier on top.

Back to the zipping up.

Two and a half minutes ago, Arizona had this thing done and dusted. Now they needed a clutch shot to tie the game up and give themselves a chance. They didnt get it. Allen got free in the paint for a shot that wasnt horrible, but he wasnt able to convert and Xavier continued its second half trend by not allowing the Wildcats a second look at the rim.

One possession for the whole thing. With Xavier in a 2-3, Parker Jackson-Cartwright set a screen on Quentin Goodin to try to free Trier at the top of the key. Goodin - the strongest guard on the Xavier roster - fought over the screen in time to close out hard on Trier. Arizonas leading scorer jabbed stepped, used a dribble to create space, and lifted from deep. Q challenged high and hard, but - like Remy Abell a year ago - pulled his hands back to avoid contact. This time it rimmed out, Mal grabbed the board, and Xavier celebrated.

I cant find exactly where, but earlier this year I questioned if the Zip Em Up era was over at Xavier. Myles Davis represented a strong link to that time, but hes gone. Instead of burying games in the second half, X was fading out of them. It was all going to seed there in February.

Then something flipped a switch. Im sure part of it was Malcom Bernard, part of it was Coach Mack, and part of it was just each guy deciding what he wanted to be. Whatever it was, it all came together last night. In the six biggest defensive possessions of the year, Xavier held a top 20 offense to 0-5/0-2/0-0 shooting, forced a turnover, and gave up zero offensive rebounds. It was man, it was zone, it was a total team effort. It was everything we needed it to be.

White sheets. Yellow tape. Closed case.

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Anatomy of a zipping up - Banners On The Parkway

Anatomy

16th-century book ‘may offer clues’ to female anatomy knowledge lag – Times of Malta

A censored 16th-century anatomy book may provide evidence that taboos slowed the development of knowledge of the female genitals, researchers have said.

The 1559 edition of Thomas Geminis Compediosa Totius Anatomie Delineatio features a depiction of a semi-dissected female torso, and the books original owner has cut away a neat triangle of paper on which the vagina would have been drawn.

It will be displayed in an exhibition at St Johns College at the University of Cambridge, and curator Shelley Hughes said it may offer clues as to why knowledge of the female anatomy lagged behind that of the human body as a whole.

She said the books original owner was disturbed by its depiction of a semi-dissected female torso.

We know this because the offending part, a neat triangle of paper on which the vagina would have been drawn, has been carefully cut away.

Sin and female flesh were held in close association in 16th-century society

She continued: Sin and female flesh were held in close association in 16th-century society with naked women often portrayed as the servants of Satan.

Before the 16th century, many European academics believed that female genital organs were simply lesser versions of male organs, turned inside out.

This dated back to classical medical authorities such as Galen in the 2nd century, who had been prohibited by law in Ancient Rome from cutting up human corpses.

The 16th century was a time of medical revolution, with pioneering researchers such as Andreas Vesalius challenging accepted views on anatomy, with evidence gathered from human dissections and direct observation experiment.

But there was still a reluctance to take on some foundational beliefs in science.

The display shows how an evidence-based knowledge of the structure of the body emerged as superstitious and religious barriers weakened.

The exhibition, on display at St Johns College at the University of Cambridge, is called Under the Knife at St Johns: A Medical History of Disease and Dissection.

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16th-century book 'may offer clues' to female anatomy knowledge lag - Times of Malta

Neuroscience

NeuroScience founder sentenced and fined for fraud | Local News … – Madison.com

The head of a western Wisconsin laboratory has been sentenced, fined and harshly admonished after being found guilty of conspiring to defraud the federal government.

Gottfried Kellermann, 76, of Osceola, was sentenced Thursday by U.S. District Judge James D. Peterson to a six-month period of home confinement, a $50,000 fine, and five years of probation for intentionally violating Clinical Laboratory Improvement Amendments regulations. Kellermans co-defendant, NeuroScience, was sentenced to a five-year probation period and a $140,000 fine for conspiring to defraud, according to the U.S. Department of Justice.

Kellermann is the founder and CEO of NeuroScience and its sister company Pharmasan Labs. In the sentencing, the court found that Kellermann was "fundamentally unrepentant" and that his allocution showed "he was a self-deluded charlatan" and that the public needed to be protected, according to a press release from the Justice Department.

According to the court, Pharmasan conducted neurotransmitter testing, and NeuroScience recommended nutritional supplements to Pharmasan patients based on the results of the testing. When Pharmasan's neurotransmitter testing did not produce consistent results for patients, Kellermann manipulated the results unbeknownst to federal regulators and patients. NeuroScience then recommended nutritional products to the patients identified as abnormal but the Justice Department said the optimal range was not valid and was "significantly narrower than the range required by federal regulators." Kellermann and his companies, who were convicted in October, hid the range and that fact it was not valid from federal regulators and from their patients.

In 2015, Pharmasan was ordered to pay the federal government $8.5 million to settle claims that it submitted false billing information to Medicare. Federal prosecutors said the settlement resolved allegations that Pharmasan and its billing company, NeuroScience, also violated Medicare rules on services referred by practitioners who weren't doctors.

Kellermann, according to the Amery Free Press, is a native of Germany who has lived in the U.S. since the 1970s. His companies have been based in Oceola, a village along the St. Croix River in Polk County, since 2002.

Originally posted here:
NeuroScience founder sentenced and fined for fraud | Local News ... - Madison.com

Neuroscience

Architects look to neuroscience to create happier, less distracting offices – HR Dive

Dive Brief:

Geek Wire reports that office design has been based on buildings first and people second. Design that puts people first can create work environments that make employees more comfortable, creative, productive and generally happier.

It's not the first time researchers have recommended experimenting with color in the workplace. One London-based research firm studied the working habits of so-called "thinking" workers in order to determine the effects of workplace stimuli. Among their findings? Cool colors and LED lights can improve alertness, and scents like lavender and jasmine can produce a calming effect.

The trend toward open, wall-free workspaces doesnt accommodate the workstyles of all employees. Hacker Moon reported on an anonymous study of 1,000 high-performing employees, many of whom are problem-solvers. The results showed that these workers reject open workspacesand instead prefer private, calm spaces where they can concentrate.

Also, as Geek Wire reported, Edward O. Wilson, Harvard University biologist and researcher, said that people are naturally attracted to open spaces.But in nature, if a predator threatens, or if in the workplace a conference call is too loud, people will retreat to a smaller, safer or quieter space.

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Architects look to neuroscience to create happier, less distracting offices - HR Dive

Neuroscience

Neuroscience: Coffee Can Protect Your Brain Against Alzheimer’s – Inc.com

Love coffee? You might be in luck. Although the fresh, aromatic bean has long been lauded for its many uses and benefits, it can be difficult to see a real use for coffee outside of its primary function -- keeping us awake when we don't have the energy to ourselves.

However, if you're an avid drinker, you might have a better understanding of the many other benefits of a cup of coffee, such as its ability to reduce heart disease, improve digestion, or even serve as a diuretic. And, in a recent study conducted by Indiana University, 24 compounds were revealed to be able to reduce the impact of harmful proteins in the brain that cause dementia.

Among these compounds, caffeine was confirmed to work alongside a powerful enzyme in the brain that allowed for the creation of a "chemical blockade against the debilitating effects of neurodegenerative diseases," said Hui-Chen Lu, a leading researcher for the study.

One of the causes of Alzheimer's is through the mis-folding -- or, for those unfamiliar with cellular processes, the improper formation -- of proteins. The targeted enzyme, NMNAT2 in this case, protects neurons from stress -- which can cause degradation as well as protein mis-folding -- thus directly combating one of the causes of neurodegenerative disease.

To determine which compounds best assisted the function of NMNAT2, more than 1,200 compounds were analyzed. And only after a large number of rigorous tests was it found that 24 compounds--including caffeine--were able to combat the negative protein mis-folding through NMNAT2 solidification. Another relatively common compound easily accessible by the general public was retinoic acid, a chemical associated with antiaging effects and cellular regeneration.

Thus, it turns out that, if you're a big caffeine addict, it might actually be able to help you more than hurt you -- provided that you're consuming responsibly, of course. If you're looking for ways to reduce your chances of harmful neurodegenerative pathways in the brain, it might be worth trying out things you normally wouldn't to better prepare your body for the inevitable long term.

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Neuroscience: Coffee Can Protect Your Brain Against Alzheimer's - Inc.com

Neuroscience

Dr. Computer is transforming neuroscience research – McGill Reporter

Browse > Home / Blog / Dr. Computer is transforming neuroscience research

Posted on Thursday, March 23, 2017

Using thousands of images from brain scans such as MRI, computers can learn to detect signs of neurological disease, opening up new possibilities in research and diagnosis.

By Shawn Hayward

In an article published in Nature on Feb. 15, researchers, including principal investigators from the Montreal Neurological Institutes McConnell Brain Imaging Centre (BIC), used magnetic resonance imaging (MRI) to predict the development of autism in babies.

It was not a neurologist or medical doctor doing the predicting, however, but a computer trained to distinguish the brains of children at risk of autism. This was an application of deep learning, a form of artificial intelligence that will increasingly put computers in the drivers seat of medical diagnosis and neuroscience research.

Developed from the concept of artificial neural networks in the 1960s, in the footsteps of the pioneering work of Donald Hebb, a former McGill psychology professor, deep learning has experienced a kind of renaissance in recent years, thanks to the increasing availability of powerful computational resources and access to vast amounts of digital data.

Deep learning involves training computers to make complex calculations after analyzing enough data to learn or detect certain patterns of interest. They do this via relatively simple algorithms that mimic the brains basic mechanisms for processing information.

If you are on Facebook, you probably have already experienced AI in action. Facebook can detect where faces are in images and will ask you if you want to tag that person. The program that makes this possible is called DeepFace, a deep learning application Facebook developed by training computers to recognize faces using four million photos manually tagged and uploaded by users.

Deep learning techniques are being used in many aspects of biomedical research. One objective is to develop computer-assisted techniques to improve diagnosis and prevention, by analyzing data of various kinds to see problems before they occur. Deep learning is particularly important to neuroscience, where data types are extremely diverse. Artificial intelligence is a promising tool to help neuroscientists discover new basic principles within the vast amount of data available.

The Nature article is just an example of how deep learning and other AI techniques are rapidly becoming important to medicine and medical research, among other fields affecting our daily lives.

Several labs at the Montreal Neurological Institute are already using deep learning and related AI techniques to conduct research, and the BIC is training the next generation of neuroscientists and brain imagers to use these new methods. In January of 2017, the BIC sponsored two hands-on educational sessions focusing on deep-learning for neuroimaging. The event was attended by 80 of the centres students and staff scientists.

AI techniques are changing the game of how we do science. We want our research staff and trainees to be aware of and well prepared for this revolution, says Sylvain Baillet, a McGill professor and Director of the BIC. We are fortunate that Montreal is emerging as an international hub for AI research and industry. To remain leaders in our field, we must embrace AI methods like deep learning together with building and using large neurodata repositories, and invest both human and technical resources to exploit the unique features of these powerful tools.

Category: Blog

Tag: autism, brain imaging, deep learning, neuroimaging, neuroscience

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Dr. Computer is transforming neuroscience research - McGill Reporter

Neuroscience

Neuroscience reveals 4 rituals that will make you stress-free – The Week Magazine

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The modern world seems to be designed to increase stress, and I'm starting to wonder if worrying will soon be an Olympic sport.

You may have your own ways of coping with stress. Problem is, research says they probably don't work.

From The Willpower Instinct:

The APA's national survey on stress found that the most commonly used strategies were also rated as highly ineffective by the same people who reported using them. For example, only 16 percent of people who eat to reduce stress report that it actually helps them. Another study found that women are most likely to eat chocolate when they are feeling anxious or depressed, but the only reliable change in mood they experience from their drug of choice is an increase in guilt. [The Willpower Instinct]

So let's go after this stress thing where it lives: your brain. There are some great methods to train your mind to reduce stress.

But they take work. And right now you're too stressed out for any of that. (Or maybe you're just lazy and impatient. Hey, I don't judge.)

So we need some stuff that's diabolically easy and backed by neuroscience research but let's keep the emphasis on diabolical. If your brain won't play fair, neither will we. So what do we need here?

Old-fashioned treachery. Of the neuroscience variety. Time to do an end run around your brain's stress response and exploit physiology to trick it into calming down. Let's play neurological hardball.

1. Clench your facial muscles and relax them

Communication between your brain and your body is a two-way street. There's a feedback loop. So if you can't get your brain to make your body calm down, you can use your body to make your brain calm down.

Your grey matter gets stressed and your muscles tighten up. Then your tense muscles send a signal back to your brain, confirming you're stressed. We gotta break the loop.

Clench your facial muscles and then relax them. Now your body is sending a signal to your brain saying, "We're not stressed anymore. You shouldn't be either."

From The Upward Spiral: Using Neuroscience to Reverse the Course of Depression, One Small Change at a Time:

To remind your brain to relax your muscles, sometimes it's helpful to clench them first. Take a deep breath in and then flex a tight muscle for a few seconds. After holding for a few seconds, exhale with a sigh and relax. The most important muscles to relax are your facial muscles, since those have the largest effect on emotion, but relaxing your hands, butt, and stomach are also important. [The Upward Spiral]

If your partner is around and you don't feel like making a face that looks like you're constipated, have them give you a massage. That works too.

(To learn the four rituals neuroscience says will make you happier, click here.)

So funny faces can beat stress. Or maybe you're getting a massage instead. That's even better. You're less stressed and you're bonding with your partner.

But what if facial scrunching doesn't work? What other dirty physiological tricks do we have?

2. Take slow, deep breaths

The vagus nerve is one of the key emotional highways in your body. It sends signals down to your heart and up to your brain playing a critical role in regulating the fight-or-flight system.

Directly stimulating the vagus nerve can fix all your issues. Only problem is that would require a scalpel and a lot of medical school loans. So we'll stay focused on treachery.

How you breathe can hijack the way the vagus nerve works. In fact, it's one of the fastest ways to change your emotional state.

From The Upward Spiral: Using Neuroscience to Reverse the Course of Depression, One Small Change at a Time:

Breathing affects the brain through signals carried by the vagus nerve. Not only does the vagus nerve send signals down to the heart, as mentioned earlier, but it also carries signals up into the brain stem. Vagus nerve signaling is important in activating circuits for resting and relaxation, known as the parasympathetic nervous system. The parasympathetic system is the opposite of the sympathetic nervous system, which controls the fight-or-flight instinct. Slow breathing increases activity in the vagus nerve and pushes the brain toward parasympathetic activity. So slow, deep breathing calms you down. [The Upward Spiral]

So how do you do it right?

Breathe in slowly through your nose while counting slowly to six (or even eight). Pause for a couple seconds at the top of your inhalation and then exhale slowly through your nose for the same count. [The Upward Spiral]

And this is no small effect. After the U.S. military taught Navy SEAL recruits a few psychological tricks (including proper breathing) passing rates jumped from 25 percent to 33 percent.

An interesting side note: Do the opposite and you'll get the opposite effect. Need more energy? Breathe quickly.

By contrast, rapid breathing deactivates the parasympathetic nervous system and activates the sympathetic nervous system. When you are anxious, excited, or scared, you breathe quickly. But it's also true that if you breathe quickly, you're more likely to feel those feelings. Fast breathing can make you more nervous but also more excited. Sometimes that's a good thing. Maybe you need a bit more energy to make it to the gym (or to do anything at all). Try quick, shallow breaths for 20 to 30 seconds. [The Upward Spiral]

(For more from neuroscientist Alex Korb on how to make your brain happy, click here.)

What if this doesn't work? You're huffing and puffing and you're still worried your house is going to get blown down. Head to the sink, my friend...

3. Splash your face with cold water

Cold water on your face will jolt your vagus nerve and slow your heart rate. Your brain feels your heart rate dropping and says, "We must not be stressed anymore." (Ha! Stupid brain)

From The Upward Spiral: Using Neuroscience to Reverse the Course of Depression, One Small Change at a Time:

Sudden cold water on your face slows down your heart rate by indirectly stimulating the vagus nerve. If you're feeling overwhelmed, stressed, or anxious, find a sink, fill your hands with cold water, and splash it on your face. [The Upward Spiral]

(To learn the methods bomb disposal experts use to stay calm under pressure, click here.)

Alright, maybe you've made funny faces, you're breathing like Darth Vader and your face is soaking wet but you're still stressed. Do not worry. (Or I should say, "Do not worry even more.") Neuroscience has another sneaky trick. And this one is fun.

4. Play music and do a little dance

Music affects how you feel, right? Fight the bad feelings with good feelings by listening to the music you love.

Sound overly simple? Nope. Your favorite song will passively help straighten out key limbic system regions like your hippocampus, your anterior cingulate, and your nucleus accumbens. Making music has an even more powerful effect.

And, no, you don't have to do a little dance. But you get bonus points if you do.

From The Upward Spiral: Using Neuroscience to Reverse the Course of Depression, One Small Change at a Time:

Whether playing an instrument or listening to the radio, music increases heart-rate variability, though making music has a stronger effect. Music engages most of the limbic system, including the hippocampus, anterior cingulate, and nucleus accumbens, which is why it can be motivating and enjoyable and can help regulate your emotions. It can also be soothing, lowering blood pressure and reducing stress. So sing along with the radio or just make a playlist of your favorite songs. Better yet, go dancing. Dancing combines music, exercise, and being social, so you get a triple boost to an upward spiral. [The Upward Spiral]

(To learn what ancient wisdom says will make you happy, click here.)

Alright, you've fooled your brain into a state of calm that would make Zen masters envious. Let's round up what we learned and do something fun together that will relieve stress and make you smile instantly

Sum up

Here's how neuroscience and treachery can make you stress-free:

So how can you kill stress and be happier with almost no effort whatsoever?

Research shows that owning a dog reduces stress. In fact, the effect is so powerful that just watching a video of a cute animal reduces heart rate and blood pressure in under a minute.

From 59 Seconds: Change Your Life in Under a Minute:

In an innovative study, Deborah Wells examined whether merely looking at a video of an animal can have the same type of calming and restorative effects as those created by being in its company Compared to the two control conditions, all three animal videos made the participants feel much more relaxed. To help reduce your heart rate and blood pressure in less than a minute, go online and watch a video of a cute animal. [59 Seconds]

You want easy stress relief? All you have to do is click

(Awwwww, that's so sweet I think I'm getting cavities.)

Join 250K+ readers. Get a free weekly update via email here.

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Neuroscience reveals 4 rituals that will make you stress-free - The Week Magazine

Immunology

Immunology Conference | Immunology Meeting 2017 | Malaysia | Asia

Scientific Sessions

Session Tracks

Conference Series invites all the participants from all over the world to attend"9th Annual Meeting on Immunology and Immunologist, July 03-04, 2017 Kuala Lumpur,Malaysiaincludesprompt keynote presentations, Oral talks, Poster presentations and Exhibitions.

TheImmunology conferencesdeals with the major branches like Classical immunology, Clinical immunology, Osteoimmunology,Medicine immunology, Tissue-based immunology. It will broadly classify the cells of immune system, autoimmune diseases, antigen-antibody reactions, T cell development, B cell development, cytokines etc. Immunology has applications in numerous disciplines of medicine, particularly in the fields of organ transplantation,oncology, virology, bacteriology, parasitology, psychiatry, and dermatology. Immunologists employed by universities work in virtually every life science department or division conducting research to increase our understanding of the immune system.

Track:1Cellular Immunology

The study of the molecular and cellular components that comprise the immune system, including their function and interaction, is the central science ofimmunology. The immune system has been divided into a more primitive innate immune system and, in vertebrates, an acquired oradaptive immune system

The field concerning the interactions among cells and molecules of the immunesystem,and how such interactions contribute to the recognition and elimination of pathogens. Humans possess a range of non-specific mechanical and biochemical defences against routinely encountered bacteria, parasites, viruses, and fungi. The skin, for example, is an effective physical barrier to infection. Basic chemical defences are also present in blood, saliva, and tears, and on mucous membranes. True protection stems from the host's ability to mount responses targeted to specific organisms, and to retain a form of memory that results in a rapid, efficient response to a given organism upon a repeat encounter. This more formal sense of immunity, termed adaptive immunity, depends upon the coordinated activities of cells and molecules of the immune system.

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 2Inflammatory/Autoimmune Diseases

Autoimmune diseasescan affect almost any part of the body, including the heart, brain, nerves, muscles, skin, eyes, joints, lungs, kidneys, glands, the digestive tract, and blood vessels.

The classic sign of an autoimmune disease is inflammation, which can cause redness, heat, pain, and swelling. How an autoimmune disease affects you depends on what part of the body is targeted. If the disease affects the joints, as inrheumatoid arthritis, you might have joint pain, stiffness, and loss of function. If it affects the thyroid, as in Graves disease and thyroiditis, it might cause tiredness, weight gain, and muscle aches. If it attacks the skin, as it does in scleroderma/systemic sclerosis, vitiligo, andsystemic lupus erythematosus(SLE), it can cause rashes, blisters, and colour changes. Many autoimmune diseases dont restrict themselves to one part of the body. For example, SLE can affect the skin, joints, kidneys, heart, nerves, blood vessels, and more. Type 1 diabetes can affect your glands, eyes, kidneys, muscles, and more.

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 3T-Cells and B-Cells

T cell: A type of white blood cell that is of key importance to the immune system and is at the core of adaptive immunity, the system that tailors the body's immune response to specific pathogens. The T cells are like soldiers who search out and destroy the targeted invaders. Immature T cells (termed T-stem cells) migrate to the thymus gland in the neck, where they mature and differentiate into various types of mature T cells and become active in the immune system in response to a hormone called thymosin and other factors. T-cells that are potentially activated against the body's own tissues are normally killed or changed ("down-regulated") during this maturational process.There are several different types of mature T cells. Not all of their functions are known. T cells can produce substances called cytokines such as the interleukins which further stimulate the immune response. T-cell activation is measured as a way to assess the health of patients withHIV/AIDSand less frequently in other disorders. T cell are also known as T lymphocytes. The "T" stands for "thymus" -- the organ in which these cells mature. As opposed to B cells which mature in the bone marrow.B cells, also known asBlymphocytes, are a type of white bloodcellof the lymphocyte subtype. They function in thehumoral immunitycomponent of the adaptive immune system by secreting antibodies. Many B cells mature into what are called plasma cells that produce antibodies (proteins) necessary to fight off infections while other B cells mature into memory B cells. All of the plasma cells descended from a single B cell produce the same antibody which is directed against the antigen that stimulated it to mature. The same principle holds with memory B cells. Thus, all of the plasma cells and memory cells "remember" the stimulus that led to their formation. The maturation of B cells takes place in birds in an organ called the bursa of Fabricus. B cells in mammals mature largely in the bone marrow. The B cell, or B lymphocyte, is thus an immunologically important cell. It is not thymus-dependent, has a short lifespan, and is responsible for the production ofimmunoglobulins.It expresses immunoglobulins on its surface.

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology Germany

.http://annualmeeting.conferenceseries.com/immunologist/

Track: 4Cancer and Tumor Immunobiology

The tumour is an important aspect of cancer biology that contributes to tumour initiation, tumour progression and responses to therapy. Cells and molecules of the immune system are a fundamental component of the tumour microenvironment. Importantly,therapeutic strategies for cancer treatmentcan harness the immune system to specifically target tumour cells and this is particularly appealing owing to the possibility of inducing tumour-specific immunological memory, which might cause long-lasting regression and prevent relapse in cancer patients.The composition and characteristics of the tumour microenvironment vary widely and are important in determining the anti-tumour immune response.Immunotherapyis a new class ofcancer treatmentthat works to harness the innate powers of the immune system to fight cancer. Because of the immune system's unique properties, these therapies may hold greater potential than current treatment approaches to fight cancer more powerfully, to offer longer-term protection against the disease, to come with fewer side effects, and to benefit more patients with more cancer

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 5 Vaccines

A vaccine is a biological preparation that improves immunity to a particular disease. A vaccine typically contains an agent that resembles a disease-causing microorganism, and is often made from weakened or killed forms of the microbe, its toxins or one of its surface proteins. The agent stimulates the body's immune system to recognize the agent as foreign, destroy it, and "remember" it, so that the immune system can more easily recognize and destroy any of these microorganisms that it later encounters. There are two basictypes of vaccines: live attenuated and inactivated. The characteristics of live and inactivatedvaccinesare different, and these characteristics determine how thevaccineis used. Liveattenuatedvaccinesare produced by modifying a disease-producing (wild) virus or bacteria in a laboratory.

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 6Immunotherapy

Immunotherapy,also called biologic therapy, is a type of cancer treatment designed to boost the body's natural defences to fight the cancer. It uses materials either made by the body or in a laboratory to improve, target, or restore immune system function. Immunotherapy is treatment that uses certain parts of a persons immune system to fight diseases such as cancer. This can be done in a couple of ways:1)Stimulating your own immune system to work harder or smarter to attack cancer cells2)Giving you immune system components, such as man-made immune system proteins. Some types of immunotherapy are also sometimes called biologic therapy or biotherapy.

In the last few decadesimmunotherapyhas become an important part of treating some types of cancer. Newer types of immune treatments are now being studied, and theyll impact how we treat cancer in the future. Immunotherapy includes treatments that work in different ways. Some boost the bodys immune system in a very general way. Others help train the immune system to attack cancer cells specifically. Immunotherapy works better for some types of cancer than for others. Its used by itself for some of these cancers, but for others it seems to work better when used with other types of treatment.

Many different types of immunotherapy are used to treat cancer. They include:Monoclonal antibodies,Adoptive cell transfer,Cytokines, Treatment Vaccines, BCG,

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 7Neuro Immunology

Neuroimmunology, a branch of immunologythat deals especially with the inter relationships of the nervous system and immune responses andautoimmune disorders. It deals with particularly fundamental and appliedneurobiology,meetings onneurology,neuropathology, neurochemistry,neurovirology, neuroendocrinology, neuromuscular research,neuropharmacologyand psychology, which involve either immunologic methodology (e.g. immunocytochemistry) or fundamental immunology (e.g. antibody and lymphocyte assays).

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 8Infectious Diseases and Immune System

Infectious diseases are caused by pathogenic microorganisms, such as bacteria, viruses, parasites or fungi; the diseases can be spread, directly or indirectly, from one person to another.Zoonotic diseasesare infectious diseases of animals that can cause disease when transmitted to humans. Some infectious diseases can be passed from person to person. Some are transmitted by bites from insects or animals. And others are acquired by ingesting contaminated food or water or being exposed to organisms in the environment. Signs and symptoms vary depending on the organism causing the infection, but often include fever and fatigue. Mild complaints may respond to rest and home remedies, while some life-threatening infections may require hospitalization.

Many infectious diseases, such as measles andchickenpox, can be prevented by vaccines. Frequent and thorough hand-washing also helps protect you from infectious diseases

There are four main kinds of germs:

Bacteria - one-celled germs that multiply quickly and may release chemicals which can make you sick

Viruses - capsules that contain genetic material, and use your own cells to multiply

Fungi - primitive plants, like mushrooms or mildew

Protozoa - one-celled animals that use other living things for food and a place to live

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 9Reproductive Immunology,

Reproductive immunologyrefers to a field of medicine that studies interactions (or the absence of them) between the immune system and components related to thereproductivesystem, such as maternal immune tolerance towards the fetus, orimmunologicalinteractions across the blood-testis barrier. The immune system refers to all parts of the body that work to defend it against harmful enemies. In people with immunological fertility problems their body identifies part of reproductive function as an enemy and sendsNatural Killer (NK) cellsto attack. A healthy immune response would only identify an enemy correctly and attack only foreign invaders such as a virus, parasite, bacteria, ect.

The concept of reproductive immunology is not widely accepted by all physicians.Those patients who have had repeated miscarriages and multiple failed IVF's find themselves exploring it's possibilities as the reason. With an increased amount of success among treating any potential immunological factors, the idea of reproductive immunology can no longer be overlooked.The failure to conceive is often due to immunologic problems that can lead to very early rejection of the embryo, often before the pregnancy can be detected by even the most sensitive tests. Women can often produce perfectly healthy embryos that are lost through repeated "mini miscarriages." This most commonly occurs in women who have conditions such asendometriosis, an under-active thyroid gland or in cases of so called "unexplained infertility." It has been estimated that an immune factor may be involved in up to 20% of couples with otherwiseunexplained infertility. These are all conditions where abnormalities of the womans immune system may play an important role.

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track:10Auto Immunity,

Autoimmunityis the system ofimmuneresponses of an organism against its own cells and tissues. Any disease that results from such an aberrantimmuneresponse is termed an autoimmune disease.

Autoimmunity is present to some extent in everyone and is usually harmless. However, autoimmunity can cause a broad range of human illnesses, known collectively as autoimmune diseases. Autoimmune diseases occur when there is progression from benign autoimmunity to pathogenicautoimmunity. This progression is determined by genetic influences as well as environmental triggers. Autoimmunity is evidenced by the presence of autoantibodies (antibodies directed against the person who produced them) and T cells that are reactive with host antigens.

Autoimmune disorders

An autoimmune disorder occurs whenthe bodys immune systemattacks and destroys healthy body tissue by mistake. There are more than 80 types of autoimmune disorders.

Causes

The white blood cells in the bodys immune system help protect against harmful substances. Examples include bacteria, viruses,toxins,cancercells, and blood and tissue from outside the body. These substances contain antigens. The immune system producesantibodiesagainst these antigens that enable it to destroy these harmful substances. When you have an autoimmune disorder, your immune system does not distinguish between healthy tissue and antigens. As a result, the body sets off a reaction that destroys normal tissues. The exact cause of autoimmune disorders is unknown. One theory is that some microorganisms (such as bacteria or viruses) or drugs may trigger changes that confuse the immune system. This may happen more often in people who have genes that make them more prone toautoimmune disorders.

An autoimmune disorder may result in:

The destruction of body tissue

Abnormal growth of an organ

Changes in organ function

A person may have more than one autoimmune disorder at the same time. Common autoimmune disorders include:

Addison's disease

Celiac disease - sprue(gluten-sensitive enteropathy)

Dermatomyositis

Graves' disease

Hashimoto's thyroiditis

Multiple sclerosis

Myasthenia gravis

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 11Costimmulatory pathways in multiple sclerosis

Costimulatory moleculescan be categorized based either on their functional attributes or on their structure. The costimulatory molecules discussed in this review will be divided into (1)positive costimulatory pathways:promoting T cell activation, survival and/or differentiation; (2)negative costimulatory pathways:antagonizing TCR signalling and suppressing T cell activation; (3) as third group we will discuss themembers of the TIM family, a rather new family of cell surface molecules involved in the regulation of T cell differentiation and Treg function.Costimulatory pathways have a critical role in the regulation of alloreactivity. A complex network of positive and negative pathways regulates T cell responses. Blocking costimulation improves allograft survival in rodents and non-human primates. The costimulation blocker belatacept is being developed asimmunosuppressivedruginrenal transplantation.

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 12Autoimmunity and Therapathies

Autoimmunityis the system ofimmuneresponsesof an organism against its own cells and tissues. Any disease that results from such an aberrantimmuneresponse is termed an autoimmune disease.

Autoimmunity is present to some extent in everyone and is usually harmless. However, autoimmunity can cause a broad range of human illnesses, known collectively as autoimmune diseases.Autoimmune diseasesoccur when there is progression from benign autoimmunity to pathogenic autoimmunity. This progression is determined by genetic influences as well as environmental triggers. Autoimmunity is evidenced by the presence of autoantibodies (antibodies directed against the person who produced them) and T cells that are reactive with host antigens.

Current treatments for allergic and autoimmune disease treat disease symptoms or depend on non-specific immune suppression. Treatment would be improved greatly by targeting the fundamental cause of the disease, that is the loss of tolerance to an otherwise innocuous antigen in allergy or self-antigen in autoimmune disease (AID). Much has been learned about the mechanisms of peripheral tolerance in recent years. We now appreciate that antigen presenting cells (APC) may be either immunogenic or tolerogenic, depending on their location, environmental cues and activation state

Related:Immunology Conferences|Immunologists Meetings|ConferenceSeries Ltd

2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

Track: 13DiagnosticImmunology

Diagnostic Immunology. Immunoassays are laboratory techniques based on the detection of antibody production in response to foreign antigens. Antibodies, part of the humoral immune response, are involved in pathogen detection and neutralization.

Diagnostic immunology has considerably advanced due to the development of automated methods.New technology takes into account saving samples, reagents, and reducing cost.The future of diagnosticimmunologyfaces challenges in the vaccination field for protection against HIV and asanti-cancer therapy. Modern immunology relies heavily on the use of antibodies as highly specific laboratory reagents. The diagnosis of infectious diseases, the successful outcome of transfusions and transplantations, and the availability of biochemical and hematologic assays with extraordinary specificity and sensitivity capabilities all attest to the value of antibody detection.Immunologic methods are used in the treatment and prevention ofinfectious diseasesand in the large number of immune-mediated diseases. Advances in diagnostic immunology are largely driven by instrumentation, automation, and the implementation of less complex and more standardized procedures.

Examples of such processes are as follows:

miniaturization (use of microtiter plates to save samples and reagents),

amplified immunoassays (chemiluminesent ELISA),

flow cytometry with monoclonal antibodies,

Immunoglobulins,

Molecular methods (polymerase chain reactions).

These methods have facilitated the performance of tests and have greatly expanded the information that can be developed by a clinical laboratory. The tests are now used for clinical diagnosis and the monitoring of therapies and patient responses. Immunology is a relatively young science and there is still so much to discover. Immunologists work in many different disease areas today that include allergy, autoimmunity, immunodeficiency, transplantation, and cancer.

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2nd InternationalConference on Antibodiesand Therapeutics, July 11-12, 2016 Philadelphia, Pennsylvania, USA; 5thEuropean Immunology Conferences, July 21-23, 2016 Berlin, Germany; 7th InternationalConference on Allergy, Asthma and Clinical Immunology, September 14-15, 2016 Amsterdam, Netherlands; 2nd internationalconference on innate immunity, July 21-22, 2016, Germany; InternationalConference on Autoimmunity, October 13-14, 2016 Manchester, UK;Immunology 2016, American Association of Immunologists, Annual MeetingMay 13-17, Los Angeles, USA Immunology Conferences;InternationalConference onMucosalImmunology, July 28-29, 2016, Australia;International Congress of Immunology

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Immunology Conference | Immunology Meeting 2017 | Malaysia | Asia

Human Behavior

True Crime Novella Highlights Human Factor in Cyber Solutions (Video) – 107.180.56.147 (press release) (registration) (blog)

ideas42, a leading non-profit behavioral design firm, hasunveiled a unique approach to highlighting and resolving key behavioral problems in cybersecurity with the launch of an original, serialized novella.

The new true-crime- style short story Deep Thought: A Cybersecurity Story dramatizes the human factors in cybersecurity and is followed by a robust index of key insights from behavioral science that can be used to improve security protocols.

The narrative, to be released in multiple installments, highlights the human actions and decisions that often compromise digital information and computer security.

These range from password issues to more complex concerns such as coding practices and organizations resource investment choices.

(Experts estimate that 70-80% of the costs attributed to cyber attacks are actually the result of human error. Thats why ideas42 is applying a behavioral science lens to what has traditionally been considered a technological problem. Courtesy ofideas42 and YouTube)

Despite public and private sector investments in sophisticated security systems, the level of risk is immense.

In the search for answers, efforts have been heavily skewed toward finding technological solutions. However, up to 80% of the cost attributed to cyber-attacks is actually a result of human error.

With Internet access rapidly expanding across the globe and the proliferation of greater connectedness across business, finance, and individuals, ensuring privacy and security is more important than ever, as underscored by recent high-profile breaches such as the hacking of American political party systems during the 2016 election cycle.

It is because of the urgency around strengthening cybersecurity that we chose to present our insights as an engaging novella instead of using the more traditional white paper approach, said ideas42 Executive Director Josh Wright.

With the release of a unique piece like Deep Thought: A Cybersecurity Story and our supporting analysis, we hope to reach more leaders and decision-makers who can take needed steps to increase the strength of their organizations digital networks.

The first installment of ideas42s novella debuted today at New Americas Cybersecurity for a New America conference and can be read at ideas42.org/cyber.

Simply clicking on a bad link can be devastating to network security, and the strongest security network in the world is only as good as the human with the password, continued Wright.

Furthermore, human error in security is not limited to end-users. The challenges around understanding and addressing human behavioral factors in cybersecurity present a rich vein of opportunity for making the system as a whole more robust.

(Hear from the author,Josh Wright, Executive Director at ideas42 on Changing the World with Behavioral Science, courtesy ofBehaviourWorks Australiaand YouTube)

ideas42s work in cybersecurity is supported by the William and Flora Hewlett Foundation Cyber Initiative in partnership with New Americas Cybersecurity Initiative. The goal is to focus on behavioral insights and solutions that can be adopted quickly and brought to scale.

For a full copy of the novella and behavioral insight appendix contact us at cyber@ideas42.org.

To use our unique experience at the forefront of behavioral science to change millions of lives.

We create innovative solutions to tough problems in economic mobility, health, education, consumer finance, energy efficiency and international development.

Our approach is based on a deep understanding of human behavior and why people make the decisions they do. Working closely with our partners from government, foundations, NGOs and companies, we have more than 80 active projects in the United States and around the world.

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True Crime Novella Highlights Human Factor in Cyber Solutions (Video) - 107.180.56.147 (press release) (registration) (blog)

Human Behavior

GU bill seeks increase in retirement age – NYOOOZ

Porvorim: The Goa University (amendment) Bill, 2017, which seeks to enhance the retirement age of the teaching staff of Goa Medical College (GMC), Goa Dental College and the Institute of Psychiatry and Human Behavior (IPHP), Bambolim, from 62 years to 65 years, was tabled in the Goa legislative assembly on Thursday.The bill proposes to amend section 15A of the Goa University Act in a bid to prevent the loss of senior and experienced teaching faculty.The GMC its...

News Source: http://timesofindia.indiatimes.com/city/goa/gu-bill-seeks-increase-in-retirement-age/articleshow/57800318.cms

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GU bill seeks increase in retirement age - NYOOOZ